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primary antibodies against b7-h4  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc primary antibodies against b7-h4
    Primary Antibodies Against B7 H4, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibodies+against+b7-h4/anti+b7+h4/pm34848831-61-25-30
    Average 90 stars, based on 1 article reviews
    primary antibodies against b7-h4 - by Bioz Stars, 2026-10
    90/100 stars

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    other:

    Article Title: Expression of B7 family checkpoint proteins in cervical cancer.
    Article Snippet: The role of programmed cell death-ligand 1 (PD-L1) in cervical cancer has been widely investigated; however, the influences of other inhibitory B7 family members are poorly understood.. We investigated the expression of PD-L1, B7 homolog 3 (B7-H3), B7-H4, and V-domain Ig suppressor of T-cell activation (VISTA) and their association with the clinicopathological features and outcomes of a large cohort of 673 patients with squamous cell carcinoma or adenocarcinoma of the uterine cervix.. The positivity rates for PD-L1 (combined positive score ≥1), B7-H3 in tumor cells (TCs), B7-H4 (exclusively in TCs), VISTA in immune cells (ICs), and VISTA in TCs were 57.9%, 62.8%, 44.8%, 92.6%, and 4.8%, respectively, in 606 primary cervical cancer samples.



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    primary antibodies against b7-h4  (GeneTex)
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    <t>B7-H4</t> expression, and CD4 + T, CD8 + T and CD11b + macrophage cell infiltration in esophagi of mice, as estimated by immunohistochemistry staining. B7-H4 was expressed in the membrane and cytoplasm of epithelial cells in the mucosa layer. Infiltration of CD4 + T, CD8 + T and CD11b + macrophage cells was predominantly located in the submucosa of esophageal tissues. Magnification, ×400. The arrows indicate the cells that are positive. B7-H4, B7-homolog 4; CD, cluster of differentiation.
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    B7-H4 expression, and CD4 + T, CD8 + T and CD11b + macrophage cell infiltration in esophagi of mice, as estimated by immunohistochemistry staining. B7-H4 was expressed in the membrane and cytoplasm of epithelial cells in the mucosa layer. Infiltration of CD4 + T, CD8 + T and CD11b + macrophage cells was predominantly located in the submucosa of esophageal tissues. Magnification, ×400. The arrows indicate the cells that are positive. B7-H4, B7-homolog 4; CD, cluster of differentiation.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: B7-H4 expression, and CD4 + T, CD8 + T and CD11b + macrophage cell infiltration in esophagi of mice, as estimated by immunohistochemistry staining. B7-H4 was expressed in the membrane and cytoplasm of epithelial cells in the mucosa layer. Infiltration of CD4 + T, CD8 + T and CD11b + macrophage cells was predominantly located in the submucosa of esophageal tissues. Magnification, ×400. The arrows indicate the cells that are positive. B7-H4, B7-homolog 4; CD, cluster of differentiation.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Expressing, Immunohistochemistry, Staining, Membrane

    Association between B7-H4 expression and pathological stage or lymphocytes infiltration in the epithelia of esophageal tissues during the carcinogenesis process.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: Association between B7-H4 expression and pathological stage or lymphocytes infiltration in the epithelia of esophageal tissues during the carcinogenesis process.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Expressing

    Gene expression of B7-H4 and associated molecules in control mice. The expression of genes in the esophagi of control mice did not change obviously from weeks 16 to 28 (B7-H4: P=0.0650; IL-6: P=0.2842; IL-10: P=0.2816; TGF-β, P=0.0743; IFN-γ: P=0.3399; STAT3: P=0.3225; Kruskal-Wallis analysis). mRNA, messenger RNA; Cq, quantification cycle; B7-H4, B7-homolog 4; CD, cluster of differentiation; IL, interleukin; TGF, transforming growth factor; IFN, interferon; STAT3, signal transducer and activator of transcription 3.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: Gene expression of B7-H4 and associated molecules in control mice. The expression of genes in the esophagi of control mice did not change obviously from weeks 16 to 28 (B7-H4: P=0.0650; IL-6: P=0.2842; IL-10: P=0.2816; TGF-β, P=0.0743; IFN-γ: P=0.3399; STAT3: P=0.3225; Kruskal-Wallis analysis). mRNA, messenger RNA; Cq, quantification cycle; B7-H4, B7-homolog 4; CD, cluster of differentiation; IL, interleukin; TGF, transforming growth factor; IFN, interferon; STAT3, signal transducer and activator of transcription 3.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Gene Expression, Control, Expressing

    Gene expression of B7-H4, IL-6, IL-10, TGF-β and STAT3 in the esophagi of 4NQO model mice increased during the carcinogenesis process (*P<0.05, **P<0.01, vs. the control group; Kruskal-Wallis analysis). mRNA, messenger RNA; Cq, quantification cycle; B7-H4, B7-homolog 4; CD, cluster of differentiation; IL, interleukin; TGF, transforming growth factor; IFN, interferon; STAT3, signal transducer and activator of transcription 3; 4NQO, 4-nitroquinoline-1-oxide.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: Gene expression of B7-H4, IL-6, IL-10, TGF-β and STAT3 in the esophagi of 4NQO model mice increased during the carcinogenesis process (*P<0.05, **P<0.01, vs. the control group; Kruskal-Wallis analysis). mRNA, messenger RNA; Cq, quantification cycle; B7-H4, B7-homolog 4; CD, cluster of differentiation; IL, interleukin; TGF, transforming growth factor; IFN, interferon; STAT3, signal transducer and activator of transcription 3; 4NQO, 4-nitroquinoline-1-oxide.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Gene Expression, Control

    Expression of B7-H4 and associated proteins in control mice. Protein expression in the esophagi of control mice did not change obviously from weeks 16 to 28 (all P>0.05, Kruskal-Wallis analysis). B7-H4, B7-homolog 4; IL, interleukin; STAT3, signal transducer and activator of transcription 3; p-, phosphorylated.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: Expression of B7-H4 and associated proteins in control mice. Protein expression in the esophagi of control mice did not change obviously from weeks 16 to 28 (all P>0.05, Kruskal-Wallis analysis). B7-H4, B7-homolog 4; IL, interleukin; STAT3, signal transducer and activator of transcription 3; p-, phosphorylated.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Expressing, Control

    B7-H4, p-STAT3 and IL-6 protein expression in the esophagi of 4NQO model mice increased during the carcinogenesis process (*P<0.05, **P<0.01 vs. the control group; Kruskal-Wallis analysis). B7-H4, B7-homolog 4; IL, interleukin; STAT3, signal transducer and activator of transcription 3; p-, phosphorylated; 4NQO, 4-nitroquinoline-1-oxide.

    Journal: Oncology Letters

    Article Title: Increased B7-H4 expression during esophageal squamous cell carcinogenesis is associated with IL-6/STAT3 signaling pathway activation in mice

    doi: 10.3892/ol.2017.5688

    Figure Lengend Snippet: B7-H4, p-STAT3 and IL-6 protein expression in the esophagi of 4NQO model mice increased during the carcinogenesis process (*P<0.05, **P<0.01 vs. the control group; Kruskal-Wallis analysis). B7-H4, B7-homolog 4; IL, interleukin; STAT3, signal transducer and activator of transcription 3; p-, phosphorylated; 4NQO, 4-nitroquinoline-1-oxide.

    Article Snippet: Sections were incubated with primary antibodies against B7-H4 (1:500; GTX62090; GeneTex, Inc., Irvine, CA, USA), CD4 (1:50; orb4241; Biorbyt, Ltd., Cambridge, UK), CD8 (1:50; orb10325; Biorbyt, Ltd.) or CD11b (1:100; cb1211; Abcam, Shanghai, China) at 4°C overnight.

    Techniques: Expressing, Control